Phenomena and Processes

T-Cell Senescence

Permanent G1 CELL CYCLE ARREST of the T-cell, resulting from ageing, oncogene activation, telomere degradation, epigenomic changes, or exposure to stressful stimuli including reactive-oxygen species, ionizing radiation, or certain chemicals.

National Library of MedicineMedical Subject Headings2026

Structured Summary

Abstract

Permanent G1 CELL CYCLE ARREST of the T-cell, resulting from ageing, oncogene activation, telomere degradation, epigenomic changes, or exposure to stressful stimuli including reactive-oxygen species, ionizing radiation, or certain chemicals.

MeSH Record

Classification

Related Concepts

Knowledge Graph

Loading graph…

Drag nodes to rearrange; hover to trace links; click a node to open its page.

MeSH Record

See Also

MeSH Record

Synonyms

14 entry terms
  • T-Cell Ageing
  • T-Cell Aging
  • T-Lymphocyte Aging
  • T-Lymphocyte Senescence
  • Ageing, T-Cell
  • Aging, T-Cell
  • Aging, T-Lymphocyte
  • Senescence, T-Cell
  • Senescence, T-Lymphocyte
  • T Cell Ageing
  • T Cell Aging
  • T Cell Senescence
  • T Lymphocyte Aging
  • T Lymphocyte Senescence

MeSH Record

Aspects Covered

5 allowable subheadings

Indexed with the subheadings drug effects, genetics, immunology, physiology, radiation effects.

MeSH Record

History Note

2024

MeSH Record

Previous Indexing

  • Cellular Senescence (1997-2023)

MeSH Hierarchy

Tree Numbers

AMA Style

References

  1. National Library of Medicine. T-Cell Senescence. Medical Subject Headings (MeSH). 2026. Unique ID D000095843. http://id.nlm.nih.gov/mesh/2026/D000095843
  2. T-Cell Senescence. In: Wikidata. https://www.wikidata.org/wiki/Q124256103