Structured Summary
Abstract
A group of PROTEIN SERINE-THREONINE KINASES which activate critical signaling cascades in double strand breaks, APOPTOSIS, and GENOTOXIC STRESS such as ionizing ultraviolet A light, thereby acting as a DNA damage sensor. These proteins play a role in a wide range of signaling mechanisms in cell cycle control.
MeSH Record
Classification
Broader headings
Related Concepts
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MeSH Record
Synonyms
7 entry terms
- A-T Protein
- AT Mutated Protein
- ATM Protein
- A T Protein
- Mutated Protein, AT
- Protein, A-T
- Protein, AT Mutated
MeSH Record
Aspects Covered
29 allowable subheadings
Indexed with the subheadings administration & dosage, adverse effects, analysis, antagonists & inhibitors, biosynthesis, blood, cerebrospinal fluid, chemical synthesis, chemistry, classification, deficiency, drug effects, economics, genetics, history, immunology, isolation & purification, metabolism, pharmacokinetics, pharmacology, physiology, poisoning, radiation effects, standards, supply & distribution, therapeutic use, toxicity, ultrastructure, urine.
MeSH Record
History Note
2014
MeSH Hierarchy
Tree Numbers
AMA Style
References
- National Library of Medicine. Ataxia Telangiectasia Mutated Proteins. Medical Subject Headings (MeSH). 2026. Unique ID D064007. http://id.nlm.nih.gov/mesh/2026/D064007
- Ataxia Telangiectasia Mutated Proteins. In: Wikidata. https://www.wikidata.org/wiki/Q24773792